rabbit anti p35 25 Search Results


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Santa Cruz Biotechnology rabbit anti p35 p25 c 19
Rabbit Anti P35 P25 C 19, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Cell Signaling Technology Inc france p35 p2587 35 25 kda rabbit anti p35 p25
France P35 P2587 35 25 Kda Rabbit Anti P35 P25, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Rabbit Polyclonal Anti Casp9 P35 P10, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Becton Dickinson rabbit anti p35 antibody
Surgical stress elevated GR phosphorylation at ser220 by CDK5 and its regulator in prefrontal cortex of POCD mice. (A), GR phosphorylation was tested with Western blot. The phosphorylation at ser220 site was significantly elevated in POCD group (two‐tailed unpaired t‐test, P = 0.0136 for pGRs220; P = 0.2303 for pGRs212; P = 0.3860 for pGRs234), compared with control group. (B) The level of CDK5 and its regulators <t>p35,</t> p25 in prefrontal cortex was tested with Western blot in POCD and control mice. The level of CDK5 was upregulated in POCD group compared with control (two‐tailed unpaired t‐test, P = 0.0178). CDK5 regulators p35 (P = 0.0140), p25 (P = 0.0139) also elevated in prefrontal cortex compared with control. (C), nonparametric correlation analysis of GR phosphorylation and behavioral index in 20‐month groups (CDK5 with MWM, r2 = 0.75, P < 0.001; CDK5 with EPM, r2 = 0.78, P < 0.001; pGRs220 with MWM, r2 = 0.63, P < 0.001; pGRs220 with EPM, r2 = 0.73, P < 0.001) (C). (*P < 0.05, POCD compared with control) (n = 8 mice/group). Error bar represents SEM. (D), no significant changes on pGRs220 (P = 0.7576), CDK5 (P = 0.8640) and its regulators (P = 0.8182 for p35 and P = 0.8844 for p25) in 6‐month groups. (*P < 0.05, POCD compared with control) (n = 8 mice/group). Data are Mean ± SEM (Error bars).
Rabbit Anti P35 Antibody, supplied by Becton Dickinson, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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BioCarta rabbit anti-p35 polyclonal antibody
Surgical stress elevated GR phosphorylation at ser220 by CDK5 and its regulator in prefrontal cortex of POCD mice. (A), GR phosphorylation was tested with Western blot. The phosphorylation at ser220 site was significantly elevated in POCD group (two‐tailed unpaired t‐test, P = 0.0136 for pGRs220; P = 0.2303 for pGRs212; P = 0.3860 for pGRs234), compared with control group. (B) The level of CDK5 and its regulators <t>p35,</t> p25 in prefrontal cortex was tested with Western blot in POCD and control mice. The level of CDK5 was upregulated in POCD group compared with control (two‐tailed unpaired t‐test, P = 0.0178). CDK5 regulators p35 (P = 0.0140), p25 (P = 0.0139) also elevated in prefrontal cortex compared with control. (C), nonparametric correlation analysis of GR phosphorylation and behavioral index in 20‐month groups (CDK5 with MWM, r2 = 0.75, P < 0.001; CDK5 with EPM, r2 = 0.78, P < 0.001; pGRs220 with MWM, r2 = 0.63, P < 0.001; pGRs220 with EPM, r2 = 0.73, P < 0.001) (C). (*P < 0.05, POCD compared with control) (n = 8 mice/group). Error bar represents SEM. (D), no significant changes on pGRs220 (P = 0.7576), CDK5 (P = 0.8640) and its regulators (P = 0.8182 for p35 and P = 0.8844 for p25) in 6‐month groups. (*P < 0.05, POCD compared with control) (n = 8 mice/group). Data are Mean ± SEM (Error bars).
Rabbit Anti P35 Polyclonal Antibody, supplied by BioCarta, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Boster Bio pcmv p35
Surgical stress elevated GR phosphorylation at ser220 by CDK5 and its regulator in prefrontal cortex of POCD mice. (A), GR phosphorylation was tested with Western blot. The phosphorylation at ser220 site was significantly elevated in POCD group (two‐tailed unpaired t‐test, P = 0.0136 for pGRs220; P = 0.2303 for pGRs212; P = 0.3860 for pGRs234), compared with control group. (B) The level of CDK5 and its regulators <t>p35,</t> p25 in prefrontal cortex was tested with Western blot in POCD and control mice. The level of CDK5 was upregulated in POCD group compared with control (two‐tailed unpaired t‐test, P = 0.0178). CDK5 regulators p35 (P = 0.0140), p25 (P = 0.0139) also elevated in prefrontal cortex compared with control. (C), nonparametric correlation analysis of GR phosphorylation and behavioral index in 20‐month groups (CDK5 with MWM, r2 = 0.75, P < 0.001; CDK5 with EPM, r2 = 0.78, P < 0.001; pGRs220 with MWM, r2 = 0.63, P < 0.001; pGRs220 with EPM, r2 = 0.73, P < 0.001) (C). (*P < 0.05, POCD compared with control) (n = 8 mice/group). Error bar represents SEM. (D), no significant changes on pGRs220 (P = 0.7576), CDK5 (P = 0.8640) and its regulators (P = 0.8182 for p35 and P = 0.8844 for p25) in 6‐month groups. (*P < 0.05, POCD compared with control) (n = 8 mice/group). Data are Mean ± SEM (Error bars).
Pcmv P35, supplied by Boster Bio, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Santa Cruz Biotechnology mouse anti il12a anti p35 antibodies
Surgical stress elevated GR phosphorylation at ser220 by CDK5 and its regulator in prefrontal cortex of POCD mice. (A), GR phosphorylation was tested with Western blot. The phosphorylation at ser220 site was significantly elevated in POCD group (two‐tailed unpaired t‐test, P = 0.0136 for pGRs220; P = 0.2303 for pGRs212; P = 0.3860 for pGRs234), compared with control group. (B) The level of CDK5 and its regulators <t>p35,</t> p25 in prefrontal cortex was tested with Western blot in POCD and control mice. The level of CDK5 was upregulated in POCD group compared with control (two‐tailed unpaired t‐test, P = 0.0178). CDK5 regulators p35 (P = 0.0140), p25 (P = 0.0139) also elevated in prefrontal cortex compared with control. (C), nonparametric correlation analysis of GR phosphorylation and behavioral index in 20‐month groups (CDK5 with MWM, r2 = 0.75, P < 0.001; CDK5 with EPM, r2 = 0.78, P < 0.001; pGRs220 with MWM, r2 = 0.63, P < 0.001; pGRs220 with EPM, r2 = 0.73, P < 0.001) (C). (*P < 0.05, POCD compared with control) (n = 8 mice/group). Error bar represents SEM. (D), no significant changes on pGRs220 (P = 0.7576), CDK5 (P = 0.8640) and its regulators (P = 0.8182 for p35 and P = 0.8844 for p25) in 6‐month groups. (*P < 0.05, POCD compared with control) (n = 8 mice/group). Data are Mean ± SEM (Error bars).
Mouse Anti Il12a Anti P35 Antibodies, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Promega anti-βiii tubulin mouse clone g7121 (1:1000)
Surgical stress elevated GR phosphorylation at ser220 by CDK5 and its regulator in prefrontal cortex of POCD mice. (A), GR phosphorylation was tested with Western blot. The phosphorylation at ser220 site was significantly elevated in POCD group (two‐tailed unpaired t‐test, P = 0.0136 for pGRs220; P = 0.2303 for pGRs212; P = 0.3860 for pGRs234), compared with control group. (B) The level of CDK5 and its regulators <t>p35,</t> p25 in prefrontal cortex was tested with Western blot in POCD and control mice. The level of CDK5 was upregulated in POCD group compared with control (two‐tailed unpaired t‐test, P = 0.0178). CDK5 regulators p35 (P = 0.0140), p25 (P = 0.0139) also elevated in prefrontal cortex compared with control. (C), nonparametric correlation analysis of GR phosphorylation and behavioral index in 20‐month groups (CDK5 with MWM, r2 = 0.75, P < 0.001; CDK5 with EPM, r2 = 0.78, P < 0.001; pGRs220 with MWM, r2 = 0.63, P < 0.001; pGRs220 with EPM, r2 = 0.73, P < 0.001) (C). (*P < 0.05, POCD compared with control) (n = 8 mice/group). Error bar represents SEM. (D), no significant changes on pGRs220 (P = 0.7576), CDK5 (P = 0.8640) and its regulators (P = 0.8182 for p35 and P = 0.8844 for p25) in 6‐month groups. (*P < 0.05, POCD compared with control) (n = 8 mice/group). Data are Mean ± SEM (Error bars).
Anti βiii Tubulin Mouse Clone G7121 (1:1000), supplied by Promega, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Santa Cruz Biotechnology anti caspase 9 p35
Surgical stress elevated GR phosphorylation at ser220 by CDK5 and its regulator in prefrontal cortex of POCD mice. (A), GR phosphorylation was tested with Western blot. The phosphorylation at ser220 site was significantly elevated in POCD group (two‐tailed unpaired t‐test, P = 0.0136 for pGRs220; P = 0.2303 for pGRs212; P = 0.3860 for pGRs234), compared with control group. (B) The level of CDK5 and its regulators <t>p35,</t> p25 in prefrontal cortex was tested with Western blot in POCD and control mice. The level of CDK5 was upregulated in POCD group compared with control (two‐tailed unpaired t‐test, P = 0.0178). CDK5 regulators p35 (P = 0.0140), p25 (P = 0.0139) also elevated in prefrontal cortex compared with control. (C), nonparametric correlation analysis of GR phosphorylation and behavioral index in 20‐month groups (CDK5 with MWM, r2 = 0.75, P < 0.001; CDK5 with EPM, r2 = 0.78, P < 0.001; pGRs220 with MWM, r2 = 0.63, P < 0.001; pGRs220 with EPM, r2 = 0.73, P < 0.001) (C). (*P < 0.05, POCD compared with control) (n = 8 mice/group). Error bar represents SEM. (D), no significant changes on pGRs220 (P = 0.7576), CDK5 (P = 0.8640) and its regulators (P = 0.8182 for p35 and P = 0.8844 for p25) in 6‐month groups. (*P < 0.05, POCD compared with control) (n = 8 mice/group). Data are Mean ± SEM (Error bars).
Anti Caspase 9 P35, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Bio-Techne corporation mouse nephrin antibody
Surgical stress elevated GR phosphorylation at ser220 by CDK5 and its regulator in prefrontal cortex of POCD mice. (A), GR phosphorylation was tested with Western blot. The phosphorylation at ser220 site was significantly elevated in POCD group (two‐tailed unpaired t‐test, P = 0.0136 for pGRs220; P = 0.2303 for pGRs212; P = 0.3860 for pGRs234), compared with control group. (B) The level of CDK5 and its regulators <t>p35,</t> p25 in prefrontal cortex was tested with Western blot in POCD and control mice. The level of CDK5 was upregulated in POCD group compared with control (two‐tailed unpaired t‐test, P = 0.0178). CDK5 regulators p35 (P = 0.0140), p25 (P = 0.0139) also elevated in prefrontal cortex compared with control. (C), nonparametric correlation analysis of GR phosphorylation and behavioral index in 20‐month groups (CDK5 with MWM, r2 = 0.75, P < 0.001; CDK5 with EPM, r2 = 0.78, P < 0.001; pGRs220 with MWM, r2 = 0.63, P < 0.001; pGRs220 with EPM, r2 = 0.73, P < 0.001) (C). (*P < 0.05, POCD compared with control) (n = 8 mice/group). Error bar represents SEM. (D), no significant changes on pGRs220 (P = 0.7576), CDK5 (P = 0.8640) and its regulators (P = 0.8182 for p35 and P = 0.8844 for p25) in 6‐month groups. (*P < 0.05, POCD compared with control) (n = 8 mice/group). Data are Mean ± SEM (Error bars).
Mouse Nephrin Antibody, supplied by Bio-Techne corporation, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Santa Cruz Biotechnology polyclonal rabbit anti podocin antibody p35
Surgical stress elevated GR phosphorylation at ser220 by CDK5 and its regulator in prefrontal cortex of POCD mice. (A), GR phosphorylation was tested with Western blot. The phosphorylation at ser220 site was significantly elevated in POCD group (two‐tailed unpaired t‐test, P = 0.0136 for pGRs220; P = 0.2303 for pGRs212; P = 0.3860 for pGRs234), compared with control group. (B) The level of CDK5 and its regulators <t>p35,</t> p25 in prefrontal cortex was tested with Western blot in POCD and control mice. The level of CDK5 was upregulated in POCD group compared with control (two‐tailed unpaired t‐test, P = 0.0178). CDK5 regulators p35 (P = 0.0140), p25 (P = 0.0139) also elevated in prefrontal cortex compared with control. (C), nonparametric correlation analysis of GR phosphorylation and behavioral index in 20‐month groups (CDK5 with MWM, r2 = 0.75, P < 0.001; CDK5 with EPM, r2 = 0.78, P < 0.001; pGRs220 with MWM, r2 = 0.63, P < 0.001; pGRs220 with EPM, r2 = 0.73, P < 0.001) (C). (*P < 0.05, POCD compared with control) (n = 8 mice/group). Error bar represents SEM. (D), no significant changes on pGRs220 (P = 0.7576), CDK5 (P = 0.8640) and its regulators (P = 0.8182 for p35 and P = 0.8844 for p25) in 6‐month groups. (*P < 0.05, POCD compared with control) (n = 8 mice/group). Data are Mean ± SEM (Error bars).
Polyclonal Rabbit Anti Podocin Antibody P35, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Surgical stress elevated GR phosphorylation at ser220 by CDK5 and its regulator in prefrontal cortex of POCD mice. (A), GR phosphorylation was tested with Western blot. The phosphorylation at ser220 site was significantly elevated in POCD group (two‐tailed unpaired t‐test, P = 0.0136 for pGRs220; P = 0.2303 for pGRs212; P = 0.3860 for pGRs234), compared with control group. (B) The level of CDK5 and its regulators p35, p25 in prefrontal cortex was tested with Western blot in POCD and control mice. The level of CDK5 was upregulated in POCD group compared with control (two‐tailed unpaired t‐test, P = 0.0178). CDK5 regulators p35 (P = 0.0140), p25 (P = 0.0139) also elevated in prefrontal cortex compared with control. (C), nonparametric correlation analysis of GR phosphorylation and behavioral index in 20‐month groups (CDK5 with MWM, r2 = 0.75, P < 0.001; CDK5 with EPM, r2 = 0.78, P < 0.001; pGRs220 with MWM, r2 = 0.63, P < 0.001; pGRs220 with EPM, r2 = 0.73, P < 0.001) (C). (*P < 0.05, POCD compared with control) (n = 8 mice/group). Error bar represents SEM. (D), no significant changes on pGRs220 (P = 0.7576), CDK5 (P = 0.8640) and its regulators (P = 0.8182 for p35 and P = 0.8844 for p25) in 6‐month groups. (*P < 0.05, POCD compared with control) (n = 8 mice/group). Data are Mean ± SEM (Error bars).

Journal: CNS Neuroscience & Therapeutics

Article Title: Surgical Stress Induces Brain‐Derived Neurotrophic Factor Reduction and Postoperative Cognitive Dysfunction Via Glucocorticoid Receptor Phosphorylation in Aged Mice

doi: 10.1111/cns.12368

Figure Lengend Snippet: Surgical stress elevated GR phosphorylation at ser220 by CDK5 and its regulator in prefrontal cortex of POCD mice. (A), GR phosphorylation was tested with Western blot. The phosphorylation at ser220 site was significantly elevated in POCD group (two‐tailed unpaired t‐test, P = 0.0136 for pGRs220; P = 0.2303 for pGRs212; P = 0.3860 for pGRs234), compared with control group. (B) The level of CDK5 and its regulators p35, p25 in prefrontal cortex was tested with Western blot in POCD and control mice. The level of CDK5 was upregulated in POCD group compared with control (two‐tailed unpaired t‐test, P = 0.0178). CDK5 regulators p35 (P = 0.0140), p25 (P = 0.0139) also elevated in prefrontal cortex compared with control. (C), nonparametric correlation analysis of GR phosphorylation and behavioral index in 20‐month groups (CDK5 with MWM, r2 = 0.75, P < 0.001; CDK5 with EPM, r2 = 0.78, P < 0.001; pGRs220 with MWM, r2 = 0.63, P < 0.001; pGRs220 with EPM, r2 = 0.73, P < 0.001) (C). (*P < 0.05, POCD compared with control) (n = 8 mice/group). Error bar represents SEM. (D), no significant changes on pGRs220 (P = 0.7576), CDK5 (P = 0.8640) and its regulators (P = 0.8182 for p35 and P = 0.8844 for p25) in 6‐month groups. (*P < 0.05, POCD compared with control) (n = 8 mice/group). Data are Mean ± SEM (Error bars).

Article Snippet: After transferred onto PVDF membrane, the following primary antibodies were used: sheep anti‐BDNF antibody (1:5000, millipore, Billerica, MA, USA); rabbit anti‐GR and anti‐p‐GRs220 antibody (1:12000, CST, Boston, MA, USA); rabbit anti‐CDK5 antibody (1:5000, BD, NJ, USA); rabbit anti‐p35 antibody (1:5000, BD); rabbit anti‐p‐GRs212 (1:2000, Anbo, San Francisco, CA, USA); and rabbit anti‐p‐GRs234 (1:12000, CST).

Techniques: Western Blot, Two Tailed Test